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Their More moderen function further revealed that the atypical GTPase area of Fbx4 could function a substrate-binding motif for that SCF E3 ligase sophisticated and bind into a globular domain of TRF1 for its ubiquitination and degradation. These conclusions provide clues for the underlying mechanism for controlling TRF1, whose expression https://emilioprojb.aioblogs.com/81979831/about-lifestyle-telomer

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