Preliminary experiments point out conolidine may possibly inhibit certain ion channels, reducing neuronal excitability and limiting suffering alerts. This mechanism is particularly relevant in neuropathic soreness, in which irregular signaling leads to persistent distress. Also, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to agony perception. Computa... https://mickm136icw2.blogs100.com/profile